In Reply to Dr. Lambein et al “HER2 protein overexpression in non‐amplified ductal carcinoma in situ: quality issue or transcription mechanisms gone awry?”

Abstract

We were pleased to read Dr. Lambein and colleagues' insightful and thought‐provoking correspondence. We acknowledge that there are alternatives to the three mechanisms we proposed to explain the discordant HER2 findings. The transcriptional mechanisms broached by Lambein et al. constitute a plausible explanation for our collective findings and are soundly supported by the literature [1‐2].

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